Staphylococcal scalded skin syndrome
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Lesea le nang le Staphylococcal scalded skin syndrome
References
Staphylococcal Scalded Skin Syndrome ke boemo boo letlalo le theohang ka lebaka la chefo e hlahisoang ke mefuta e itseng ea libaktheria tsa Staphylococcus. Ha e tloaelehe ho bana ba ka holimo ho lilemo tse tšeletseng. E ka boela ea etsahala ho batho ba baholo ba nang le tsamaiso ea 'mele ea ho itšireletsa mafung e fokolang kapa mathata a tebileng a liphio. Ntho e ka sehloohong ke ho ebola ha letlalo ka mor'a ho ruruha. Botenya bo tloha ho li-blister tse seng kae ho ea ho tahlehelo e atileng ea letlalo, e ka bakang ho theoha ho matla ha mocheso oa 'mele le ho se tsitse ha khatello ea mali.
Staphylococcal Scalded Skin Syndrome is a disease characterized by denudation of the skin caused by exotoxin producing strains of the Staphylococcus species, typically from a distant site. It usually presents 48 hours after birth and is rare in children older than six years. It may also present in immunocompromised adults or those with severe renal disease. The disorder is characterized by significant exfoliation of skin following cellulitis. The severity may vary from a few blisters to system exfoliation leading to marked hypothermia and hemodynamic instability.
Staphylococcal Scalded Skin Syndrome is a disease characterized by denudation of the skin caused by exotoxin producing strains of the Staphylococcus species, typically from a distant site. It usually presents 48 hours after birth and is rare in children older than six years. It may also present in immunocompromised adults or those with severe renal disease. The disorder is characterized by significant exfoliation of skin following cellulitis. The severity may vary from a few blisters to system exfoliation leading to marked hypothermia and hemodynamic instability.
Staphylococcal scalded skin syndrome (SSSS) and bullous impetigo are infections caused by Staphylococcus aureus. Bullous impetigo is due to the local release of these toxins and thus, often presents with localized skin findings, whereas SSSS is from the systemic spread of these toxins, resulting in a more generalized rash and severe presentation. Both conditions are treated with antibiotics that target S. aureus. These conditions can sometimes be confused with other conditions that result in superficial blistering.
Moshanyana ea likhoeli li 10 o ile a kena a ruruhile nko a sa je hantle. Lingaka li boletse hore ke ts'oaetso e holimo ea phefumoloho. Matsatsi a mabeli hamorao, o ile a khutla hobane o ne a sa ntlafala 'me a e-na le mathata a macha a kang ho ruruha sefahleho le letlalo le hlabang ka molomo. Matsatsing a mabeli a latelang, moshanyana eo o ile a mpefala le ho feta. Matsoho le maoto a hae a ruruhile mme letlalo la hae la qala ho tsoa. Ha a khutlela sepetlele, lingaka li ile tsa bona lekhopho le lefubelu sefahlehong sa hae le mamenong a letlalo la hae, le neng le soeufala ha le angoa. Ba ile ba mo fumana a na le staphylococcal scalded skin syndrome (SSSS) mme ba mo qala ka lithibela-mafu ka mothapo.
A 10-month-old male presented with rhinorrhea and decreased oral intake and was diagnosed with an upper respiratory infection. Two days later, he returned to the clinic due to a lack of improvement and the onset of new symptoms, including facial edema and perioral skin irritation. That evening, he became febrile at 100.4 °F and went to the emergency department at the local children's hospital. No further workup was done and the parents were instructed to continue with the current treatment regimen. Over the next 48 hours, the patient's symptoms worsened with the new onset of bilateral extremity edema and desquamation. The patient was returned to the emergency department. A physical exam was notable for a blanching, desquamating, erythematous rash on the face and creases of the arms, legs, and groin. A positive Nikolsky sign was reported. A clinical diagnosis of staphylococcal scalded skin syndrome (SSSS) was made, and the patient was started on intravenous clindamycin. This case illustrates a severe presentation of SSSS in a pediatric patient, demonstrating the challenges it poses to diagnosis and treatment.
A 10-month-old male presented with rhinorrhea and decreased oral intake and was diagnosed with an upper respiratory infection. Two days later, he returned to the clinic due to a lack of improvement and the onset of new symptoms, including facial edema and perioral skin irritation. That evening, he became febrile at 100.4 °F and went to the emergency department at the local children's hospital. No further workup was done and the parents were instructed to continue with the current treatment regimen. Over the next 48 hours, the patient's symptoms worsened with the new onset of bilateral extremity edema and desquamation. The patient was returned to the emergency department. A physical exam was notable for a blanching, desquamating, erythematous rash on the face and creases of the arms, legs, and groin. A positive Nikolsky sign was reported. A clinical diagnosis of staphylococcal scalded skin syndrome (SSSS) was made, and the patient was started on intravenous clindamycin. This case illustrates a severe presentation of SSSS in a pediatric patient, demonstrating the challenges it poses to diagnosis and treatment.
Ngoanana ea lilemo li 2 o ile a hlaha a e-na le lekhopho 'meleng lohle le neng le hlahile lihora tse fetang 48, kamora ho longoa ke likokoanyana sefahlehong sa hae maobane. Nakong ea tlhahlobo, o ne a e-na le lekhopho le atileng le entsoeng ka maqhubu a manyenyane a kopantsoeng hammoho, 'me ha a phunya hanyenyane, letlalo la hae le ne le bontša matšoao a letšoao la Nikolsky. Ho ne ho se na matšoao a lekhopho le amang lera la hae. Liteko tsa mali a hae ha lia ka tsa bontša matšoao leha e le afe a tšoaetso.
A 2-year-old girl presented a generalised rash with 48 h of evolution, in the context of insect bites on the face on the day before. At observation, she had a generalised micropapular rash with confluent areas and Nikolsky sign. There was no mucosal area affected. Blood cultures were negative.
A 2-year-old girl presented a generalised rash with 48 h of evolution, in the context of insect bites on the face on the day before. At observation, she had a generalised micropapular rash with confluent areas and Nikolsky sign. There was no mucosal area affected. Blood cultures were negative.
Lefu lena le bakoa ke li-epidermolytic exotoxins (exfoliatin)A le B, tse hlahisoang ke S. aureus. Tlhaloso ea staphylococcal scalded skin syndrome ho bana e ntle haholo, e na le tharollo e feletseng nakong ea matsatsi a 10 a phekolo, 'me ntle le leqeba le leholo. Leha ho le joalo, staphylococcal scalded skin syndrome e tlameha ho aroloa ka hloko ho epidermal necrolysis e chefo, e nang le bofokoli bo bobe.
○ Tlhahlobo le Kalafo
Ho hlahlojoa hampe joalo ka liso tsa eczema joalo ka atopic dermatitis, le ho sebelisoa ha setlolo sa steroid ho mpefatsa liso. Ka kopo, batla thuso ea bongaka kapele kamoo ho ka khonehang ha u ntse u sebelisa setlolo sa lithibela-mafu.
#Bacitracin
#First-generation cephalosporins (e.g. Cefradine)
#Bacterial culture
#Third-generation cephalosporins (e.g. Cefditoren Pivoxil)